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The PlA1/PlA2 polymorphism lies in the gene for that βthree (GPIIIa) subunit of αIIbβ3 (GPIIb-IIIa). This integrin serves as being a receptor for adhesive proteins fibrinogen and von Willebrand component which is important to the cohesive Homes of platelets. Recognition of those proteins as soluble ligands necessitates αIIbβ3 to generally be activated by exposing platelets to 1 of many agonists. The transition from the receptor from the resting to an activated condition depends upon inside of-out signals, which might be transmitted through the mobile membrane to render the extracellular encounter of αIIbβthree capable to bind soluble ligands.

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